C26 demethylation of brassinosteroids in arabidopsis thaliana

Cartoonish representation that illustrates the interaction between exercise and diet on the regulation of brain plasticity and cognitive function. (A) Based on experimental evidence (), exercise or a diet rich in the omega‐3 fatty acid docosahexaenoic can increase the expression of genes involved in synaptic plasticity and function while a high‐saturated fat and sucrose (HF) diet has the opposite effects. Dietary supplementation with omega‐3 fatty acids elevates levels of brain‐derived neurotrophic factor (BDNF)‐mediated synaptic plasticity in the hippocampus, a brain region important for learning and memory. Molecular changes are associated with an enhancement in hippocampal‐dependent spatial learning performance in the Morris water maze (MWM). (B) In turn, animals exposed for three weeks to a HF diet showed opposite effects to the omega‐3 fatty acid diet on BDNF levels and cognitive capacity. Concomitant exposure of the animals to voluntary running wheel exercise enhanced the effects of the omega‐3 fatty acid diet, while counteracted the effects of the HF diet on synaptic markers and cognitive ability. Values are expressed as a percentage of control (regular diet, no exercise). Modified, with permission, from reference (), p. 575

C26 demethylation of brassinosteroids in arabidopsis thaliana

c 26 demethylation of brassinosteroids in arabidopsis thaliana

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c 26 demethylation of brassinosteroids in arabidopsis thalianac 26 demethylation of brassinosteroids in arabidopsis thalianac 26 demethylation of brassinosteroids in arabidopsis thalianac 26 demethylation of brassinosteroids in arabidopsis thalianac 26 demethylation of brassinosteroids in arabidopsis thaliana

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